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Activation of AMPK has been demonstrated to suppress the expression of genes involved in DNL, whilst concomitantly upregulating those associated with FAO and lipid catabolism

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The unique ability of the BBB to sequester GSH from the blood circulation through its Na + -dependent GSH transporter provides a solid foundation for using the transporter as a target to develop brain-targeting drug delivery systems

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When GSH levels are chronically low, immune cells within the CNS shift to a damage-oriented phenotype