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As proof of effective control and modulatory effect, BPC 157 therapy effects counteracted in the same dosage range the adverse effects of NO-system disturbances, i.e., NOS-blockade (L-NAME)-induced pro-thrombotic and hypertensive effect as well as NOS-over-activity (L-arginine)-induced anti-thrombotic and hypotensive effect [126,128], and maintained thrombocytes function [128,129,130,131] also in aggregometry and thromboelastometry studies

EGF-receptor inhibition may prevent tumor cell dissemination

126,127 Glucocorticoid resistance in COPD patients may also be related to the p38MAPK pathway through phosphorylated glucocorticoid receptor (GR) reducing GR translocation into the nucleus and DNA binding, impairing the GR function

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