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The mechanism of drug resistance is that it rapidly inactivates BACH1, enabling NRF2regulated cytoprotective and detoxification genes to be induced rapidly, thus preventing AML cells from bortezomibmediated apoptosis

This article mainly focuses on mammalian Trxs, glutaredoxins, and peroxiredoxins, herein referred to as redoxins

The proteosome system (26S) is a highly selective proteolytic pathway [71]

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