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This activation leads to the stimulation of the VEGFR2-Akt-eNOS signaling pathway, enhancing endothelial tube formation and blood flow recovery in ischemic tissues

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However, it is notable that exercise was linked to the expression of several acyl-CoA dehydrogenases that were not changed with NNMTi treatment, suggesting exercised skeletal muscles shift toward -oxidation as a fuel source

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The translation of these findings requires cautious interpretation and further rigorous human studies to establish efficacy, safety, and optimal dosing