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This attenuates the accumulation of ROS and strengthens the antioxidant defense system (Zhao et al., 2017)

Acetylation strengthens the interaction between NRF2 and its transcriptional co-activators, facilitating the transcriptional activation of genes involved in antioxidant and detoxification processes

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Further experiments revealed that hydrogen-induced Nrf2 can directly bind to NF-B p65, interfering with its DNA-binding ability to pro-inflammatory gene promoters while also suppressing the release of inflammatory mediators such as HMGB1 and TNF-, thereby truncating the positive feedback loop of IKK