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J., Klein, S

monocytogenes not only for its allosteric binding to PrfA to promote expression of the virulence factors but for the ability to resist oxidative stress to produce a reductive environment for life activity (Masip et al., 2006

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P53 loss inhibits the nuclear accumulation of DPP4 and thus facilitates plasma membrane-associated DPP4-dependent lipid peroxidation, which eventually leads to ferroptosis
