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This activation of the NRF2/SLC7A11/GPX4 pathway enhanced GSH levels, decreased lipid ROS accumulation, inhibited ferroptosis and lipid peroxidation, and improved the cognitive functions of AD mice ( In summary, Ghrelin, Neuritin, GAA, and Artemisinin may mitigate cerebral neuronal injury and cognitive dysfunction by modulating neuronal ferroptosis, lipid peroxidation, the antioxidant system, oxidative stress, mitochondrial dysfunction, and neuroinflammation

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( E ) Bar plots indicating the DO enrichment for the genes involved in the tissue-sharing and tissue-specific regulation categories

[PubMed] [Google Scholar] 83.Kim H., Park J., Tak K.H., Bu S.Y., Kim E

However, the accumulation of excess MNPs in the brain may also cause toxic buildup and, thus, adverse effects