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Calcineurin-NFAT signaling controls neutrophils ability of chemoattraction upon fungal infection

This relatively high-affinity binding to MurG, as opposed to MraY, is consistent with lipid I being a natural substrate of MurG

HDLs anti-thrombotic effects are also mediated, in part, through HDLs ability to inhibit tissue factor and factors X, Va, and VIIIa (Figure 10) (832)

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98 The triad is in close proximity to unmodeled electron density which cannot be reliably assigned but might originate from Lipid II that was added into the LCP for crystallization