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These data suggest that increased GSH levels should have a positive effect on BP in hypertension, and we propose that pathological inhibition of vascular relaxation involves not only NO production by endothelial cells but also glutathione-dependent bioavailability of NO, with S-nitrosothiols playing an important role in the pathway

[117] compared the effects of aqueous and ethanol extracts of hawthorn fruit in rats with induced hyperlipidemia

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These events lead to a state of vascular inflammation, which may be partially mediated by ROS formed by activated mononuclear cells

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