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siRNA-mediated knockdown of BACH1 reversed the increased proliferation of HUVECs after BPC157 treatment (Fig

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These changes are accompanied by elevated nitric oxide levels and reduced superoxide dismutase (SOD) activity, which are indicative of oxidative stress and insulin resistance (Toskulkao et al., 1994)

Cells were differentiated in RPMI 1640 containing 25 mM HEPES, 1% penicillin/ streptomycin, 10% FBS and 20% L929 conditioned media

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