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Note, portal hypertension, along with caval and intracranial hypertension and aortal hypotension, was regularly eliminated/attenuated [129,130,131,132,133,134,135,136,137,138,139,140,141,142]

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Our results indicate that transient high glucose causes persistent OXTR suppression during subsequent normoglycemia through hyperglycemia-mediated consistent oxidative stress