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However, similar to NOX1 and NOX2, NOX4 can also be regulated by Rac1 and Akt, and protein kinase B signaling pathways (Gorin et al., 2003)

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While ferroptosis helps eliminate infected or damaged cells, its dysregulation exacerbates inflammation, tissue injury, and alveolar-capillary barrier dysfunction in ARDS ( 2.2 Key molecular mechanisms of ferroptosis Ferroptosis is characterized by the iron-dependent accumulation of lipid peroxides, driven by dysregulated oxidative stress and impaired antioxidant defenses

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doi: 10.1093/carcin/bgi267

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