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10.4049/jimmunol.1402376 65 de Arruda GrossklausD.BailoA

Herein, we focus on some of the primary lung antioxidant defenses implicated in animal models of pulmonary fibrosis and humans with IPF, including catalase, glutathione (GSH), and superoxide dismutase (SOD) as well as nuclear factor erythroid 2-related factor 2 (Nrf2), the primary regulator of antioxidant enzymes

(Peptide Sciences) Improved tissue homeostasis: Possibly improved regeneration, improved organ function, improved resilience to stress

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Activated microglia in MS and EAE are associated with the production of several pro-inflammatory cytokines (IL-6, IL-1, IL-18, IL-12, IL-23, TNF-) and chemokines (CCL2, CCL3, CCL4, CCL5, CCL7, CCL12) (135)