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Dermatology research and practice, 2020, 8547960

M., Brockmann, A., McGill, T

This paper extends prior efforts to clarify the mechanisms underlying the protective effect of dietary energy restriction on neoplastic growth, documenting changes in gene expression within tumours as a function of host diet

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1,2 Understanding the intricacies of zinc metabolism at the cellular level, including encompassing the absorption, intracellular trafficking, utilization, storage, and expulsion of zinc, can shed light on the various effects of zinc in cell physiology and pathology

In colon cancer (HCT 116) cells, emodin reduces VEGF secretion and VEGFR1/2 protein expression by down-regulating Acyl-CoA synthetase long chain family member 4 (ACSL4) [468], while in liver cancer, emodin inhibits tumorigenesis through concurrently suppressing the VEGFR2-AKT-ERK1/2 signaling pathway, and inhibiting the protein expression of SMAD2/4 by enhancing miR-34a level in Hep G2 cells [469]