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As the GSH level increases, in the more reduced intracellular environment, these bonds are disrupted and the enzyme takes on the less-active monomeric form

The dissociation of Gd (III) from Gd-chelates, which has been highlighted as the primary etiology, is more likely to occur in patients with renal dysfunction, who have a reduced excretion rate, allowing a longer retention that facilitates in vivo ion dissociation, when compared to those with normal renal function

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EGCG impacts glycolysis by the inhibition of extracellular acidification via the inhibition of both the activity and content of the glycolytic enzymes [185]