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Four alternative mechanisms might be used to explain how LC reduced TMX-induced brain injury: (1) LC directly removed ROS, suppressed brain cell lipid peroxidation, protected the cell membrane from oxidative stress, and maintained normal cell structure and functions

Key mechanisms include: Fat Oxidation: Facilitates the transport of fatty acids into the mitochondria for oxidation and conversion into usable energy, potentially enhancing fat-burning

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Glutathione can support healthy aging, protection from free radical damage, a healthy immune system and energy levels.*

Many mechanisms remain speculative, requiring further mechanistic and clinical research
