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doi: 10.1515/cclm.2003.195 27 Le BrasMClmentMVPervaizSBrennerC

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Biol Psychiatry 46:16491655

In summary, these experiments have reinforced the strategy of protecting the heart by targeting mitochondrial ion channels in early reperfusion or under other conditions of oxidative stress (e.g., during chronic cardiac disease progression)

Not only that, but often the form of B12 that these patients get is either cyanocobalamin or hydroxycobalamin

These data suggest that latency III generates lower lipid ROS levels, utilizes distinct glutathione metabolism, and/or compensatory mechanisms to control lipid ROS