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NOX-mediated aberrant ROS production in response to proinflammatory signaling is a major source of oxidative stress in epilepsy, and is further linked to interictal glucose hypometabolism (Malkov et al., 2018)

Ridder IS, Rozeboom HJ, Kalk KH, Janssen DB, Dijkstra BW (1997) Three-dimensional structure of l-2-haloacid dehalogenase from Xanthobacter autotrophicus GJ10 complexed with the substrate-analogue formate

The peptide consistently demonstrates protective effects against various forms of organ damage in animal models

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First, the specific signaling pathways or factors responsible for SIRT1 downregulation in the cochlea remain unknown

Magnesium-dependent ATPase activity and cooperativity of magnesium chelatase from Synechocystis sp