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Functionally, each pathway circumvents melanomas intrinsic apoptosis resistance, and their dysregulationvia genetic mutations, epigenetic silencing, or metabolic reprogrammingcontributes to tumor progression and therapeutic resistance, highlighting their shared role as tumor-suppressive mechanisms when functionally intact (112, 114)

Human COQ4 deficiency: delineating the clinical, metabolic and neuroimaging phenotypes

Accumulation of SOD1 mutants in postnatal motoneurons does not cause motoneuron pathology or motoneuron disease

HDL, high density lipoprotein, LDL, low density lipoprotein

Superimposition of the hTrx1 structures obtained in the hTrxR1hTrx1 complexes with published structures in the uncomplexed form (PDB codes 2hsh 18 , and 1aiu 17 ) also shows r.m.s