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Discouraging clinical read-through: The most advanced human program on the same HGF/c-Met hypothesis (a different molecule, fosgonimeton) failed its primary endpoint in the Phase 2/3 LIFT-AD Alzheimers trial [6] [7]

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The role of CD38 in the tumor microenvironment on glioma progression was also investigated by comparing WT and CD38-deficient mouse models