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10.3390/ijerph10093886 159 ValkoM.LeibfritzD.MoncolJ.CroninM

GSSG is then reduced back into GSH by the activity of glutathione reductase, which requires NADPH to regenerate the mitochondrial pool of GSH (reviewed in greater detail in Andreyev et al

In line with this, we previously found that host 1-antitrypsin can augment C

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mTORC1 and mitochondria in the regulation of cell death mTORC1 and mitochondria are involved in modulating different forms of programmed cell death, including apoptosis, ferroptosis, cuproptosis, and pyroptosis
