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Abstract Background: Oxidative stress is known to play a role in critical illness due to an imbalance in reactive oxygen species and reactive nitrogen species, and the bodys ability to detoxify pro-oxidants using small molecule anti-oxidants and anti-oxidant enzymes

In addition, the activity and expression of SLC7A11 is strictly regulated by multiple mechanisms, such as transcription factor 4 (ATF4), nuclear factor erythroid 2related factor 2 (NRF2), ubiquitin BRCA1 (Breast cancer gene 1)-associated protein-1 (BAP1), the SWI/SNF complex, and rapamycin complex 1 (mTORC1) (51), through which the expression of SLC7A11 can be upregulated to enhance the antioxidant defense of cells and inhibit the occurrence of ferroptosis ( Figure 1B )

It inhibits the ubiquitination and degradation of STAT1 by preventing the binding of the E3 ubiquitin ligase NEDD4L to STAT1 and promoting the interaction between STAT1 and the deubiquitinating enzyme USP7, thereby increasing its stability

doi: 10.1038/s41380-024-02526-4 56

ROS are highly reactive substances concentration of which depends on the physiological state of the organism

doi: 10.1074/jbc.R113.472936, 81