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The key strength of this study is that our patient population and study design are representative of a real-world clinical setting, including patients with various baseline HbA 1C concentration, weight, and BMI values who are taking other concomitant antihyperglycemic medications and have a history of prior GLP-1 RA and/or insulin use

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To confirm our observation of low B12-induced FAO impairment in AT, we assessed mitochondria functional integrity in a preadipocyte cell line and primary adipocytes, and the correlation of B12 with FAO genes in mature human scWAT and omWAT

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