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Inhibition of tobacco smoke-induced lung inflammation by a catalytic antioxidant

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In addition, the mitochondrial Ca 2+ uniporter (MCU) and mitochondrial permeability transition pore (mPTP) have been proposed as pathways for iron entry into mitochondria [87, 88], and blocking MCU by Ru360 reduced mitochondrial iron uptake and showed higher efficacy than blocking the mPTP with cyclosporin A as it leads to more profound decreases mitochondrial ROS levels and increased mitochondrial function, indicating that MCU may be a crucial portal for iron uptake by mitochondria [89]

The effects and potential toxicity of other forms remain untested

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