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Higher intracellular ROS would be expected to deplete NADPH, and since cholesterol biosynthesis depends on the NADPH availability, these findings seemed counter-intuitive

The findings were a reduction of ATP production in the cell line together with disruption of mitochondrial DNA (mtDNA) but without an increase in reactive oxygen species (ROS) or activity of caspase-3 and caspase-7, which would be the case for apoptosis (Janik-Karpinsa et al., 2022)

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doi:10.1213/ANE.0000000000001546 Donovan LM, Kapur VK

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