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Their experiments showed that ferroptotic inhibitor Fer-1 or iron chelates DXZ mitigated lipopolysaccharide (LPS)-induced ferroptotic cell death in sepsis cardiomyopathy model, while ferroptosis inducers sorafenib and erastin exacerbated LPS-induced myocardial injury

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These findings suggest that a strategy aimed at sustaining or enhancing brain GSH may be a viable approach to mitigate secondary injury processes induced by TBI

doi: 10.1016/j.heliyon.2023.e22973

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