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Conversely, mutant p53 (mtp53) promotes glycolysis through HK2 upregulation [185] and mTOR-PKM2 activation [186, 187], potentially conferring cuproptosis resistance

Its extremely short half-life restricts its diffusion capability to other parts of the cells while also enhancing its potency[4]

PMID 12036582

The method of 28

Daam2-PIP5K is a regulatory pathway for Wnt signaling and therapeutic target for remyelination in the CNS

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