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glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

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glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

Sequence alignment and data filtering The Cellranger v7.1.0 count workflow with default settings was used to process the single-cell sequencing data for subsequent analysis, facilitating data quantification, genome alignment against the GRCh38 version (sourced from 10x Genomics), and preliminary data filtration

glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

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glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

Contribution of carrier-mediated transport systems to the bloodbrain barrier as a supporting and protecting interface for the brain

glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

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glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

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glutathione crohn& 39 Gut microbiota-related metabolism is key mechanism for sulforaphane ameliorating ulcerative colitis A Th17 cell-intrinsic glutathione/mitochondrial-IL-22 axis

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