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Lv Z, Shan X, Tu Q, Wang J, Chen J, Yang Y
HCV infection in healthy CD4 + T cells blocks Top1 protein production and enzymatic activity, which results in mitochondrial dysfunction, topological mtDNA damage, and programmed cell death through numerous signaling pathways
Finlay and N
UBIAD1, a cholesterol-synthesizing enzyme, has been identified as the underlying mechanism responsible for MK-4 accumulation in tissues by catalyzing phylloquinone conversion into MK-4 [21]
L.AbateD.AbateK
Complex HLA-DR and -DQ interactions confer risk of narcolepsy-cataplexy in three ethnic groups
