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This paper reviews the Met metabolism in normal conditions and its dysregulation under abnormal conditions, the possible causes of hypermethioninemia, and its connection to BA pathogenesis: Abnormal hepatic level of Met could lead to a perturbation of redox homeostasis and mitochondrial functions of hepatocytes, enhancement of viral infectivity, and dysregulation of innate and adaptative immune cells in response to infection/damage of the liver contributing to the initiation/progression of BA

Hypertens Res 43 , 582584 (2020)

Macromol Mater Eng

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Cell migration and invasion assays Wound healing experiments and transport assays were used to assess cell migration and invasion capacity
Archived from the original on 21 March 2021