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However, the use of cisplatin as a chemotherapy agent is limited by drug resistance and side effects due to the formation of inactive metabolites from the conjugation reaction between cisplatin and Glutathione S-Transferase (GST)

Delayed production of free radicals following noise exposure

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MCF7, a breast cancer cell line which expresses high levels of PlexinB1, were treated with Sema4D, and RanGTP levels were detected with RanBP1(RBD)-GST, which binds to GTP-bound Ran specifically [39]

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