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An F, Wang H, Liu Z, Wu F, Zhang J, Tao Q, et al

[DOI] [PubMed] [Google Scholar] 824.Macfadyen AJ, Reiter C, Zhuang Y, Beckman JS

In this study, CGA improved the activity of mitochondrial complexes I, IV, and V by enhancing the levels of SOD and mitochondrial glutathione in the brain, inhibited the activation of Bax and caspase-3 (proapoptotic proteins), increased the expression of Bcl-2 (an antiapoptotic protein), and prevented the MPTP-mediated apoptotic cascade

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\n\n\n\n Importantly, BPC-157 has not been found to significantly raise or lower blood pressure in healthy animal models, and it does not appear to disrupt hormonal axes in ways that would suggest meaningful endocrine system interference

Start Understand the mechanism Use the quick facts, pathway overview, and research notes to understand why the compound is discussed
