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or saline 5.0 mL/kg i.p.) given at 5 min after isoprenaline, we assessed the early occlusion-like syndrome in the isoprenaline-treated rats as an early multiorgan failure (brain, heart, lung, liver, kidney and gastrointestinal lesions), thrombosis, intracranial (superior sagittal sinus) hypertension, portal and caval hypertension, and aortal hypotension, full presentation

Moghrabi N, Clarke DJ, Burchell B, Boxer M

185 Inconsistencies in the outcome of these studies might be attributed to the heterogeneity of the populations studied and/or different conditions of analysis and concentrations of plasma cysteine used

However, despite the many clinical applications of NO, the physiological changes depend on its concentration, which is difficult to control because of its short half-life [31]

These processes convert ethanol into the toxic intermediate acetaldehyde [2, 19]

The suggested dithiol mechanism was based on experiments at a single substrate concentration that required more than 8 h for redox equilibration in the presence of 0.5 M glutaredoxin with or without 1 M GSH