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When the gut epithelial barrier is disrupted, lipopolysaccharide (LPS) and other detrimental microbial products enter the host's circulation, leading to the activation of systemic inflammation [2]

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To this point, animal models have demonstrated rapid time courses of enhanced beta cell Ca 2+ signalling and intra-islet connectivity after bariatric surgery, before weight loss occurs, which may explain acute improvements in insulin secretion [89]