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Mechanistically, AOD9604 appears to upregulate the expression of 3-adrenergic receptors (3-AR) in adipose tissue, which are key mediators of lipolysis in fat cells

This review integrates current evidence on how brain peptides contribute to AD pathophysiology, summarizes recent progress in peptide-based therapeutic strategies and delivery platforms, and critically examines the remaining barriers to clinical translation, including bloodbrain barrier penetration, metabolic stability, off-target effects, and the need for biomarker-guided patient stratification