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Nuclear oncoprotein prothymosin is a partner of Keap1: implications for expression of oxidative stress-protecting genes
Whether TMAO is a causative factor in the development and advancement of OP or merely a biomarker of underlying pathology remains unclear
Blocking phosphorylation impairs ArfGAP1-induced neurite outgrowth inhibition in primary neurons and protects against the neurotoxic effects of PD-linked G2019S LRRK2
Controlador de temperatura automtico
Methionine dependency and cancer treatment
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