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Finding a pathogenic mutation in a RTT patient can be used to support the clinical findings in that individual

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Exogenous MT and CK together inhibit drought-induced leaf senescence by maintaining chlorophyll content (through down-regulation of chlorophyll catabolic metabolism genes), enhancing photochemical efficiency, preserving relative water content, and improving physiological traits in terms of reduced H 2 O 2 content, lipid peroxidation and electrolyte leakage

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In the present case, we found HMB45 and CKAE1-AE3 in the large cells and S100 protein, synaptophysin and NSE in the small cell component