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Ninetyone per cent of drugrelated adverse events in subjects administered ProSavin were minimal and/or manifested during a 1year posttreatment period

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Mice- and human-derived cardiac organoids exposed to different concentration of MPs demonstrated increased oxidative stress, inflammation, apoptotic cell death and collagen accumulation, which resulted associated to hypertrophic-related genes expression, such as myosin, heavy chain 7B (MYH7B), atrial natriuretic peptide (ANP), brain natriuretic peptide (BNP), Collagen, type I, alpha 1 (COL1A1) [70] ( Figure 3 )

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