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Riboflavin, vitamin B2, attenuates NLRP3, NLRC4, AIM2, and non-canonical inflammasomes by the inhibition of caspase-1 activity
IGF-1 promotes the development and cytotoxic activity of human NK cells
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The improper functioning of enzymes may result from polymorphisms in their genes

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Environmental factors may have a previously unrecognized role in the etiology of these lesions
