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This metabolic imbalance contributes to increased lipid peroxidation, as indicated by high malondialdehyde (MDA) levels ( These mitochondrial impairments not only compromise energy production but also act as pro-inflammatory stimuli, promoting chronic neuroinflammation through dysregulated mitochondrial signaling and innate immune response

(2018), however, (Round and Mazmanian, 2009)

Another extracellular Cu-Zn-dependent isoenzyme is also reported

The process of creating ATP via the electron transport chain naturally produces a small amount of free radicals