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The authors also acknowledge the support from Fundamental Research Funds for the Central Universities (Z.T., D5000210635 and Y.Q., D5000210829)

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Researchers became interested when preclinical studies demonstrated that adding glucagon receptor agonism to incretin-based therapy produced metabolic effects that exceeded those of GLP-1 or GIP activation aloneparticularly in energy expenditure and hepatic lipid metabolism

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Any consideration of this therapy must occur under strict, licensed medical supervision

Khoury A, Pagan KA, Farland MZ