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In PD models, the use of dependent sodium channel blockers reduces glutamatergic hyperactivity and inhibits abnormal glutamate release, suggesting that amino acid metabolism disturbances may contribute to neurotoxicity and oxidative toxicity, ultimately resulting in dopaminergic neuron death [85]

Free Radical Bio Med 40:21552165 Kubo A, Sano T, Saji H, Tanaka K, Kondo N, Tanaka K (1993) Primary structure and properties of glutathione-reductase from Arabidopsis thaliana

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Rituximab depletes CD20-positive B cells

Bacteriological Quality of Raw Camel Milk in the Middle Region (Sajir), Saudi Arabia. East African Scholars Journal of Medical Sciences 1: 6469

Nrf1 knockout mice die in utero but fetal hepatocytes and embryonal fibroblasts from these animals have lower GSH levels and are more susceptible to oxidative stress (Kwong et al., 1999