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Recent studies have shown that HIF-1 activation exhibits cell-specific effects in the diabetic kidney, with elevated HIF activity in glomerular cells promoting glomerulosclerosis and albuminuria, while tubular HIF activity is often suppressed, leading to mitochondrial dysfunction and tubulointerstitial fibrosis [15]

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Compartmental distribution of cellular NAD(H) and NADP(H) The intracellular distribution of NAD(H) and NADP(H) is highly compartmentalized owing to specific localization of their biosynthetic enzymes and membrane permeability to these dinucleotides (106)