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Description
Nat Rev Cardiol 17:773789
One possibility is that pink-1 and pdr-1 operate independently to regulate -synuclein toxicity in worm dopaminergic neurons and loss of each gene is responsible for an additive phenotype observed in double mutants
L.HempstockJ.SkepperJ
doi: 10.3390/ani14131868 40 PyoralaS
However, the model assumes that any changes in the plasma drug levels will always result in a relative change in tissue drug levels
[PMID: 1724477] 6
