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The use of MET inhibitors may disrupt typical downstream pathways, specifically JAK/STAT1, which are common to both MET and IFN-, thereby impeding the IFN--triggered expression of PD-L1 and PD-L2 in tumors with MET amplification

Direct in vitro Comparison of Daratumumab with Surrogate Analogs of CD38 Antibodies MOR03087, SAR650984 and Ab79

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