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Tregs can also directly inhibit natural killer (NK) cell effector functions by the down-regulation of NKG2D receptors on the surface of NK cells caused by membrane-bound TGF- on Tregs (231)
The TrxRs differ from GR by having a catalytic selenocysteine redox-active site at the C-terminus in addition to the standard CXXC domain (Lu and Holmgren, 2014)
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[DOI] [Google Scholar] 221.Cirri M., Mennini N., Maestrelli F., Mura P., Ghelardini C., Mannelli L
doi: 10.3168/jds.2012-6317 78
