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Nevertheless, there is now a consensus suggesting that the effect of Se intake on hyperglycemia has a U-shaped doseresponse curve, i.e., a dose above the recommended level (55 micrograms/day) causes hyperglycemia (and hyperinsulinemia) [55,57]

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The latter pathway requires a three-stage process, catalyzed by selenophosphate synthase organifying Se (probably in the form of Se 2 or H 2 Se) in an ATP-dependent process to activated selenophosphate, which phosphorylates seryl-tRNA synthetase (Figure 3)