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AD and MCI brain and plasma display extensive oxidative stress as indexed by protein oxidation, lipid peroxidation, ...
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Consequently, mitochondrial dysfunction exacerbates oxidative stress, impairs mitochondrial DNA (mtDNA) or deletions of mtDNA, increases intracellular Ca2+ levels, changes in fission/fusion and mitochondrial morphology, and lastly leads to neuronal death
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Its antioxidant properties help neutralize and prevent free radicals from UV rays and pollution
