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Outcomes studied included changes in sensitivity to heat, cold, and mechanical pain, as well as measures of mood and opioid withdrawal symptoms

We conclude that METH directly attenuates CREB protein level and activation through an undefined mechanism in cardiomyocytes, which might underlie METH-induced downregulation of the mitochondrial fission regulatory protein Fis1 level in cardiomyocytes

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Pathological neuroinflammatory conversion of reactive astrocytes is induced by microglia and involves chromatin remodeling

Rare but serious events encompass provocation or exacerbation of seizures in susceptible individuals and hypersensitivity reactions ranging from urticaria to anaphylaxis

Follow-up of participants continued for 28 days