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The ryanodine receptor is involved in release of Ca 2+ from the SR so, although it is evident that SELENON loss results in impairment of Ca 2+ homeostasis, the origin of this defect remains unclear and may involve both the release (ryanodine receptor) and re-uptake (SERCA) of Ca 2+

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SGs were fixed in 4% paraformaldehyde (02890-45

Phospholipids are important structural components of cell membranes, and the sphingolipid, sphingomyelin, is a component of the myelin sheath that enhances nerve transmission (29)

LTF therefore appears to be a multifaceted molecule with many positive effects on metabolism, immunity and neurological function that would be of benefit in AD